To What Extent Does the Role of Mitochondria Affect the Life and Death of Human Skin Cells? (Published)
Mitochondria are thought to be one of the most vital organelles in cellular metabolism. One key function of mitochondria is to generate the energy currency of the cell, however, there are several other ways in which mitochondria are critical to cellular survival. The objective of this article is to investigate to what extent the role of mitochondria affects the life and death of human skin cells. In terms of the methodology, the study will be carried out using qualitative research to exhibit the substantial role of mitochondria through release of free radicals, mitochondrial DNA, apoptosis and use of anti-ageing reagents. Further research could be conducted to examine the impacts of exercise, diet and environment on mitochondrial function.
Keywords: Apoptosis, anti-ageing, free radicals, mitochondria, mitochondrial DNA
Human Genome & Origin of Mitochondria (Published)
It had been believed that when individual eukaryotic cells engulfed aerobic and photosynthetic bacteria, the bacteria became endosymbionts with the phagocyte and evolved into mitochondria and chloroplasts respectively. The key objective of this study is to prove that the Endosymbiotic Theqory about the origins of mitochondria and chloroplasts is a misleading lie, being ridiculously laughable. Each of the micrographs or videos in the Figures, and each of the concrete evidences listed were designed to have targetful aims. The micrographs, videos, and concrete evidences were devised to serve as addressive findings (results). The human mitochondrial DNA is a transcript obtained when two separate spliced or mature RNAs (of DNA type) transcribed from the nuclear Genome of human oocyte have annealed into double-stranded transcript and exported into the cytoplasmic organelle called mitochondrion (plural, mitochondria) that mainly performs the task of producing energy (ATP) to power the genomic reactions of the cell. Lynn Margulis’s Endosymbiotic Theory which stated that mitochondrion & chloroplast found in eukaryotic cells had evolved from bacteria when they were engulfed by eukaryotic cells, was a misleading lie that would had been ridiculously laughable!!!!
Citation: Eriso F. (2022) Human Genome & Origin of Mitochondria, European Journal of Biology and Medical Science Research, Vol.10, No.4, pp.,33-56
Keywords: ATP, Annealing, Chloroplasts, Endosymbiotic theory, Splicing, Transcript, mitochondria, mitochondrial DNA
To What Extent Does The Role Of Mitochondria Affect The Life And Death Of Human Skin Cells? (Published)
Citation: Diyana Anthony (2022) To What Extent Does The Role Of Mitochondria Affect The Life And Death Of Human Skin Cells, European Journal of Biology and Medical Science Research, Vol.10, No.2, pp.1-7
Abstract: Mitochondria are thought to be one of the most vital organelles in cellular metabolism. One key function of mitochondria is to generate the energy currency of the cell, however, there are several other ways in which mitochondria are critical to cellular survival. The objective of this article is to investigate to what extent the role of mitochondria affects the life and death of human skin cells. In terms of the methodology, the study will be carried out using qualitative research to exhibit the substantial role of mitochondria through release of free radicals, mitochondrial DNA, apoptosis and use of anti-ageing reagents. Further research could be conducted to examine the impacts of exercise, diet and environment on mitochondrial function.
Keywords: Apoptosis, anti-ageing, free radicals, mitochondria, mitochondrial DNA
Food Intake and Caffeine Determine Amyloid Beta Metabolism with Relevance to Mitophagy in Brain Aging and Chronic Disease (Published)
In the global world diabetes and mitochondrial disease is expected to cost the developing world in the next 30 years US $400 million. In diabetes an absent peripheral sink amyloid beta clearance pathway is now relevant to amyloid beta induced mitochondrial apoptosis. The quality of food consumed has raised major concerns with increased levels of plasma bacterial lipopolysaccharides (LPS) that induces amyloid beta aggregation and mitochondrial apoptosis with programmed cell death linked to non alcoholic fatty liver disease (NAFLD) and many organ diseases. The amount, nature and time of day of fat consumption in diabetes has become important with relevance to caffeine metabolism, brain toxic amyloid beta oligomer formation and neuron apoptosis. To prevent programmed cell death dietary fat and caffeine consumption need to be revised to allow rapid hepatic caffeine and amyloid beta metabolism with the prevention of global mitophagy associated with diabetes, NAFLD and neurodegenerative diseases.
Keywords: Diabetes, amyloid beta, caffeine, circadian rhythm, fat diet, mitochondria
